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zeynep's profile
zeynep tufekci
zeynep tufekci
zeynep tufekci
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@zeynep

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zeynep tufekciVerified account

@zeynep

Complex systems, wicked problems. Society, technology, science and more. @UNC professor. @NYTimes columnist. My newsletter is @insight: http://www.theinsight.org 

floating in a most peculiar way
theinsight.org
Joined August 2009

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    1. Jasnah Kholin - 8964 - ACAB -  💉 💉‏ @wanderer_jasnah 21 Dec 2021
      Replying to @Merz @zeynep and

      yeah but those (apart from monodispersity -- but that feels like it'd require more substantial mutations in M&E to occur) are all differences in infectivity (viral particle able to enter cell & replicate) not transmission (viral particle going from point A to B).

      2 replies 0 retweets 1 like
    2. Alex Merz‏ @Merz 21 Dec 2021
      Replying to @wanderer_jasnah @zeynep and

      Physically smaller infectious units might be more susceptible to target barrier defenses. Increased resistance to those defenses could increase the relative importance of airborne transmission.

      1 reply 0 retweets 0 likes
    3. Alex Merz‏ @Merz 21 Dec 2021
      Replying to @Merz @wanderer_jasnah and

      Or rather, efficiency.

      1 reply 0 retweets 0 likes
    4. Alex Merz‏ @Merz 21 Dec 2021
      Replying to @Merz @wanderer_jasnah and

      Not written as clearly as I'd like. LMK if not clear.

      2 replies 0 retweets 0 likes
    5. Jasnah Kholin - 8964 - ACAB -  💉 💉‏ @wanderer_jasnah 21 Dec 2021
      Replying to @Merz @zeynep and

      so what i think you're saying (correct me if i'm wrong) is you propose that smaller/"aerosol-sized" particles could potentially be more likely to be inhibited by i.e. mucus/pulmonary surfactant so resistance to that would lead to more efficient aerosol infectivity?

      1 reply 0 retweets 1 like
    6. Jasnah Kholin - 8964 - ACAB -  💉 💉‏ @wanderer_jasnah 21 Dec 2021
      Replying to @wanderer_jasnah @Merz and

      thing is, i'm skeptical such a change in resistance could occur via changes in the fusion protein alone (& Omicron mutations are extremely concentrated here). that being said this would be easy to test: is there a dependence on particle size in infectivity & if yes how strong?

      2 replies 0 retweets 0 likes
    7. Jasnah Kholin - 8964 - ACAB -  💉 💉‏ @wanderer_jasnah 21 Dec 2021
      Replying to @wanderer_jasnah @Merz and

      (feels like this data should exist *somewhere* for at least D614G Wuhan-Hu-1).

      2 replies 0 retweets 1 like
    8. Jasnah Kholin - 8964 - ACAB -  💉 💉‏ @wanderer_jasnah 21 Dec 2021
      Replying to @wanderer_jasnah @Merz and

      i don't disagree such things are *possible* -- but it's far more *likely* transmission characteristic changes are due to the same things as in past variants: increased fusogenicity (this time much more cell-type dependent than before) & higher % of viable viral particles.

      2 replies 0 retweets 1 like
    9. Alex Merz‏ @Merz 21 Dec 2021
      Replying to @wanderer_jasnah @zeynep and

      I agree that those changes are important. The immune evasion alone makes previously infected or twice-vaccinated hosts humorally naive to the point that omicron might be a legit new serogroup. The cell specificity data need to be replicated.

      1 reply 0 retweets 0 likes
    10. Jasnah Kholin - 8964 - ACAB -  💉 💉‏ @wanderer_jasnah 21 Dec 2021
      Replying to @Merz @zeynep and

      agree w/you on this being a new serogroup, but in that case how much of *not* observing similar spread w/Delta *was* high seropositivity that was protective? saw some pretty vertical epi curves back then too, just burning out a lot sooner.

      1 reply 0 retweets 1 like
      zeynep tufekci‏Verified account @zeynep 21 Dec 2021
      Replying to @wanderer_jasnah @Merz and

      Yep

      9:28 PM - 21 Dec 2021
      0 replies 0 retweets 0 likes

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