Another thread on the risk of sublethal mutagenesis (on the virus, not the human!) from molnupiravir:https://twitter.com/michaelzlin/status/1464424333246447620 …
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So why don't we just skip molnupiravir and *just* do the Pfizer drug?
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One running issue is I’m not sure there is a good institutional mechanism or evidence-generating culture within the FDA’s drug or device divisions specific to the population-level risks and benefits of infectious disease treatments and test (vax transmission, rapid tests also)
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Simple question: How would this mechanism compare to 1) immune suppressed patient creation of VOCs 2) incomplete population vaccination 3) Cross-over from animals 4) spontaneous mutations.?This strikes me as theoretical when the virus has so many other significant drivers.
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The bigger problem is that it does not work particularly well and and has a narrow window of intervention. How many patients will get in to their primary care doc in time to get treated? Likely insurance preauthorization will take the full five days (sarcasm alert).
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completing a course of treatment regardless of symptoms typically leads to more resistance than stopping after symptoms resolve.https://www.who.int/news-room/questions-and-answers/item/antimicrobial-resistance-does-stopping-a-course-of-antibiotics-early-lead-to-antibiotic-resistance …
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Is anyone familiar with psychological research concerning a correct adoption of instructed medication intake schemes among people who are (or who are living in a household with) more doubting of the necessity of a medical intervention?
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@threadreaderapp unroll please -
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