Delta seems more severe on average, so would expect it to be clinically less mild on average as well for a re-infection following a non-Delta initial infection (as vaccines: Delta breakthroughs seem more severe), but same principle would hold to a Delta infection/re-infection.https://twitter.com/rrabagast/status/1463888409257201672 …
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The confusion seems to be the misunderstanding that viruses become weaker as an adaptive strategy (not true, they don’t need to: selection is on transmission, not virulence directly). OTOH, the immune system exists! So host *response* to the *same* virus can be different.
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Also, understanding conditional probability is key. Symptomatic/severe re-infections are much more likely to be detected (mild/asymptomatic ones less likely to be noticed), thus cannot be a denominator for comparison to clinical outcomes of infections. Need comprehensive surveys.
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I hold the same view as last May, when Delta become clear. This increased transmissibility means that most of the world will have encountered the spike of this virus soon enough. The question is if via vaccination or infection/exposure. Delta makes the latter is even higher risk.https://twitter.com/zeynep/status/1398253140751917062 …
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Anyway, vaccinating the world remains the same urgent issue. As few primary infections as possible is job number one. That’s not all there is, obviously, but that’s still job number one.
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What do we call a novel coronavirus when it’s not novel any more?
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A cold.
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this is pre-Delta expect worse outcomes with Deltahttps://www.medrxiv.org/content/10.1101/2021.11.23.21266767v1 …
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all pre delta?
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The population in the study was almost all under 50.
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