Since day one, @zeynep has been right about COVID. It’s easy to say big outdoor gatherings (rallies, protests, concerts) are superspreader events. (I’m guilty too.) But the data just don’t support that. Instead, it’s the everyday indoor stuff we don’t see that’s killing people.https://twitter.com/zeynep/status/1363491410075389962 …
-
-
I think, eventually, we'll learn "droplets"—emissions that travel with gravity—were minor threat from people who weren't coughing/sneezing but the floaty pieces (aerosols) were responsible for even most of the close-contact spread. Otherwise, you'd see a lot more outdoor spread.
-
With that mental model, downside: close contact is always an issue especially if unmasked talking/shouting—though seems some people can generate a lot of these floaty pieces just by breathing. Upside: anything that dilutes concentration (air/wind) or deactivates (sun) is helpful.
- Show replies
New conversation -
-
-
There was a thread from an Australian man who claimed to have caught COVID from his friend after they were drinking beers and talking across a wooden table outside at a pub. I have no way of finding that thread again though, other than asking my Aus buddies if they remember
Thanks. Twitter will use this to make your timeline better. UndoUndo
-
-
-
It’s definitely possible. If you can picture air turbulence (smoker nearby, for example)—imagine sitting at a corner table for a couple of hours eating/talking, within one foot of each other, no wind, a more sheltered aspect outside.
-
Sure. Has anyone estimated infectious viral load from a ppm/volume perspective? I realize there are countless variables. But in the same way that second hand smoke outdoors is detectable but likely not materially harmful, I’m curious about COVID (and resp infections generally).
End of conversation
New conversation -
Loading seems to be taking a while.
Twitter may be over capacity or experiencing a momentary hiccup. Try again or visit Twitter Status for more information.