I get your point about epistasis of these key variants in B.1.1.7. Do you think the N501Y alone could account for the heightened transmission?
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Because I think what he is outlining here is a very real possibility. https://twitter.com/tom_tom2021/status/1341469403762880514 …
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Does this possibility factor in as a potential drawback to focusing initial vaccinations primarily based on age vs risk of exposure? Especially since so much more of the data upon which decision making is based is available for mortality relative to morbidity?
End of conversation
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