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Interactions that promote intestinal tumor formation, cellular mechanisms of cystic fibrosis disease, and the activation of an atypical kinase in this year’s first Science Signaling issue! http://bit.ly/2BVPmn7 pic.twitter.com/AEDefXejEI
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Burkhardt lab
@CHOP_Research identifies Crk family adaptor proteins are critical for T cell migration#CellBiology http://bit.ly/2LggqBs pic.twitter.com/KIyycSQkUa
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GPR35 promotes Na/K-ATPase function to increase glycolytic metabolism and cellular proliferation
@Cambridge_Uni#Immunometabolism http://bit.ly/2R5GdCw pic.twitter.com/0o9f5tr1bT
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A polyunsaturated fatty acid activates sensory neurons and requires the GPCR Mrgprd to promote abdominal pain associated with IBS
@Inserm@Univ_Toulouse http://bit.ly/2QA5ZP8 pic.twitter.com/N7qIDzrkLg
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Yates Lab
@scrippsresearch finds the most prevalent mutation in Cystic Fibrosis patients prevents phosphorylation necessary for CFTR chloride channel maturation http://bit.ly/2R1Hlam pic.twitter.com/wEw41ZwJJD
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Want to understand synaptic plasticity in learning and memory? Don’t miss the latest Review from Johannes Hell
@UCDavisMed on AMPA receptor trafficking and postsynaptic localization#neuroscience http://bit.ly/2R9nS7S pic.twitter.com/bX9PFbM7cG
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Live-cell imaging reveals that the sphingolipid S1P activates atypical PKC to promote cell survival
@UCSDMedSchool http://bit.ly/2BYfc9Q pic.twitter.com/39u14Awovj
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Posttranslational modification of the chloride channel CFTR correlates with the cellular response to therapy
@scrippsresearch http://bit.ly/2R1Hlam pic.twitter.com/HzmR8Crn8a
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Independent of its traditional ligands, GPR35 interacts with the Na/K-ATPase pump to enhance intestinal tumor formation
@Cambridge_Uni http://bit.ly/2R5GdCw pic.twitter.com/Dh7dWBvOpq
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Covalent EGFR family inhibitors promote degradation of TRIB2 to kill cancer cells http://bit.ly/2QYInEa
@LivUni@Pseudoenzyme#pseudokinase#cancer#BestOf2018. Happy New Year's!pic.twitter.com/8pwvJa7WG3
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This year, researchers uncovered a targetable HDAC3–SMARCA4–miR-27a axis that promotes PAX3:FOXO1 fusion oncogene expression in rhabdomyosarcoma http://bit.ly/2BVxuc2
@cctdilab#HDACinhibitor#cancer#BestOf2018pic.twitter.com/4dkO4z8yVE
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Using CRISPR and siRNA, the Lefkowitz lab
@DukeMedSchool elucidated the manifold roles of β-arrestins in#GPCR signaling http://bit.ly/2BQZSfd#BestOf2018pic.twitter.com/kXSufCXxCJ
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cAMPr: A genetically encoded single-wavelength fluorescent sensor for cyclic AMP. http://bit.ly/2BT8AJR
@nyuschoolofmed@justinblau01#cellimaging#BestOf2018pic.twitter.com/95QoxdUP9C
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A research group found that hypoxia-induced interaction of filamin with Drp1 causes mitochondrial hyperfission–associated myocardial senescence. http://bit.ly/2BVo9ku
@KyushuUniv_EN#heartattack#hypoxia#BestOf2018pic.twitter.com/FIMY2kriVD
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This year, a study from
@WeillCornell discovered that inhibitors of adenylyl cyclase increase melanosome pH, skin pigmentation, and may reduce skin cancer risk: http://bit.ly/2BYVeMr#melanoma#drugrepurposing#BestOf2018pic.twitter.com/XqU0DO2ggG
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In September, a study described how the kinases HipA and HipA7 phosphorylate different substrate pools in Escherichia coli to promote multidrug tolerance http://bit.ly/2QVtgLE
@uni_tue@uni_copenhagen#antibioticresistance#BestOf2018pic.twitter.com/mqRKKjdrpZ
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Adenosine inhibits the migration of T cells from cancer patients due to reduced KCa3.1 K+ channel activity http://bit.ly/2QXnmd1
@uofcincy@ameetchimote#ionchannels#cancerimmunity#BestOf2018pic.twitter.com/ONkUVYebU6
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In our July 3 issue, a study found inhibiting the acetyltransferase NAT10 rescued a nuclear import pathway in cells from Hutchinson-Gilford progeria syndrome patients. http://bit.ly/2BNfekM
@SPJacksonGroup@GurdonInstitute#progeria#aging#BestOf2018pic.twitter.com/yztv0pLkPH
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R-Ras2 stimulates tRNA expression necessary for mitochondrial biogenesis and GC B cell development
@UAM_Madrid@pilar_daroca#immunometabolism#BestOf2018 http://bit.ly/2BJSimt pic.twitter.com/rT89GWuyG2
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As we count down to the new year, we'll be highlighting ten of our favorite research articles from 2018. Look out for tweets with the
#BestOf2018 tag as we celebrate a year of great research!pic.twitter.com/vfMxgi18iE
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