another tentative clue that antibodies are an important culprit (at least in severe disease): early indications that IL-6 is high and anti-IL-6 is beneficial. Cytokines stuff is always murky but a major function of IL-6 is to induce production of IgG antibodies
circumstantial evidence: healthcare workers have really high rates of the disease for their age. both COVID-19 and SARS, could be because they have more opportunities for exposure, could be because they have *repeated* exposure, so higher doses.
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i've seen people discussing that evidence, and the mechanism seems sensible enough. i just wonder if maybe it's swamped by a zillion other factors in real-world data? also hard to actually observe. you can measure "initial" viral load but that's usually days after infection
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