these antibodies, triggering various effector mechanisms in the lungs, induce a new round of inflammation and damage, analogous to transfusion-related acute lung injury
https://www.thelancet.com/pdfs/journals/laninf/PIIS1473-3099(20)30232-2.pdf … points to severe COVID-19 having higher viral load (also from nasopharyngeal swabs)
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oh yeah, i remember that one. don't know offhand how to explain the difference. an italian paper also says there's no clear difference in viral load btw symptomatic vs. asymptomatic https://arxiv.org/ftp/arxiv/papers/2003/2003.09320.pdf …pic.twitter.com/EH039clUxn
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and another one before i forget (too many damn papers!) https://www.sciencedirect.com/science/article/pii/S1473309920301961#fig2 …pic.twitter.com/OL0dxPAVJk
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Here's SARS: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC527336/ … initial viral load correlates to more severe disease
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circumstantial evidence: healthcare workers have really high rates of the disease for their age. both COVID-19 and SARS, could be because they have more opportunities for exposure, could be because they have *repeated* exposure, so higher doses.
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