macrocephalopod

2) cov2 has a very hard time infecting bat lung cells. It shows a huge preference towards human ace2 receptors vs other species. No other naturally found pathogen so recently introduced in humans shows this tendency.

3) cov2 has 99.5% of the amino acids required to bind to human ACE2 receptors. No other naturally occurring pathogen recently introduced to humans has ever displayed that level of optimization

Again, yellow card. Discovered. Most viruses have not been discovered.

What are the odds that an undiscovered virus is 99.5% optimized for binding to human ACE2? Ask any scientist and they will tell you it is extremely low

Weasel words and lack of context. Plus, given the density of viruses and rapid rate of recombination and mutation, quite high over time? One of the theories I read last year, the origin of the English variant, was simply an immuno-compromised patient giving a ton of time for

the virus to evolve. Depending on environmental factors, it's quite common for evolutionary optima to be reached. It's how genetic algorithms work.

Would appreciate refraining insults. You never answered the questions. What are the odds an undiscovered virus is 99.5% optimized specifically to binding to human ACE2? Are you srsly refuting that it's low probability?