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ICYMI | In this News&Views, Katharina Wolter & Lars Zender explore how cancer therapies can trigger
#senescence of tumour cells and a new study showing that eradication of therapy-induced senescent cells (#senolysis) can improve#LiverCancer therapy https://go.nature.com/30Vhzq3 pic.twitter.com/HzZtnnPIpw
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When your endothelial cells get old
#Aging#Senescence#insulinresistance@NatureCommshttps://www.nature.com/articles/s41467-020-14387-w … -
How chronic
#stress turns your#hair#grey: Hyperactivation of sympathetic nerves drives depletion of melanocyte#stem cells.#aging#ageing#senescence#grayhttps://is.gd/2i1PNd -
#TBT REVIEW | Cellular#senescence in#gastrointestinal diseases: from pathogenesis to therapeutics https://go.nature.com/2ka8okf pic.twitter.com/r2yMFwDcfw
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Great read to start the year. - CS-exposed p16-/- mice exhibit reduced emphysema, less ACECII senescence and better lung function.
#senescence https://www.nature.com/articles/s42003-019-0532-1 …Prikaži ovu nit -
We're hiring! 2 postdoc positions available
#tumor#immunology#senescence#exosomes https://www.ohsu.edu/school-of-medicine/pucci-lab … https://www.nature.com/articles/d41586-019-00350-3 …pic.twitter.com/KM7lc8w7ib
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Cardiac glycosides are broad-spectrum senolytics
#senescence at@NatMetabolismhttps://www.nature.com/articles/s42255-019-0122-z?fbclid=IwAR3VA9BG1KMqAmTtYbU9G03lQ2WFS1W4NX3M4rcmDrFBk68YRWDJs6a1iSQ … -
Two papers, published today in
@NatMetabolism and@NatureComms, identify cardiac glycosides as a new class of senolytic drugs, active in vitro and in animal models. Work from Jesus Gil@MRC_LMS and@mcollado_CHUS@fidis_research#aging#senescence#cancerhttps://www.nature.com/articles/s42255-019-0122-z …Prikaži ovu nit -
Des chercheurs américains sont parvenus à désactiver un des gènes responsables de la
#progéria, cette pathologie qui accélère la#sénescence. Ce n'est pas pour cela qu'on pourra ralentir le vieillissement ! https://www.parismatch.com/Actu/Sciences/Va-t-on-pouvoir-ralentir-le-vieillissement-1644185 … -
In a study supported by the Division of Cancer Biology, researchers
@CCLRI et al. showed that the Kindlin2-p53-SerpinB2 signaling pathway regulates cellular#senescence in#breastcancer@nresearchnews https://go.nature.com/2ZM8Fch .pic.twitter.com/UPFFRjPxve
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#Senescence : hücrelerin sessiz moda geçmesi normal ve kemoterapi görmüş hücrelerde oldukça farklı seyrediyor. Normal hücre: p21 proteini “siklin bağımlı kinazlar”ı inhibe eder ve hücre bölünmez. Kemoterapi görmüş hücre: p21 hücre bölünmesini teşvik eder https://www.nature.com/articles/d41586-019-02336-7?utm_source=twt_nv&utm_medium=social&utm_campaign=newsandviews … -
Is
#p53 signaling a driver or downstream effector of#aging? The p53-MDM2 axis is key to many aspects of both pathological and normal aging… read this beautiful review by Danyi Wu & Carol Prives on CDD@cddpress!#mousemodels#senescence https://www.nature.com/articles/cdd2017187 …pic.twitter.com/9OEnBseBjI
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“We're trying to study the reasons cells age, so that we might be able to design treatments for better aging." This researcher reflects on his investigation of
#senescence: http://ow.ly/yqaR50vbUTd -
Our AUGUST issue out NOW has all of this: therapeutic vulnerabilities of paediatric solid tumours,
#senescence, emerging roles of YAP & TAZ in the#tumourmicroenvironment & alternative tumour-specific antigens for#cancervaccine development@NatResCancer https://go.nature.com/2MhY5qm pic.twitter.com/FCG6ySuIic
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New review by my friend and colleague
#SheilaStewart#rolemodel#WomeninSTEM@SitemanCenter@WUSTLmed Unmasking#senescence : context-dependent effects of#SASP in#cancer @NatureRevCancerhttps://www.nature.com/articles/s41568-019-0156-2?utm_source=nrc_etoc&utm_medium=email&utm_campaign=toc_41568_19_8&utm_content=20190726&WT.ec_id=NRC-201908&sap-outbound-id=3DD53468B929C47492C5DC8E5EDDFF78796378BC&utm_source=hybris-campaign&utm_medium=email&utm_campaign=000_SKN6563_0000013386_41568-NRC-v19-n8-EAlert&utm_content=EN_internal_29957_20190726&mkt-key=005056A5C6311EE999B009074D4669E3 … -
Knockout of Kindlin-2 via
#CRISPR/Cas9 in several#BreastCancer cell lines significantly increased expression levels of both SerpinB2 and p21 resulting in the activation of hallmarks of cellular#senescence. | In@cddpress, by researchers from@CCLRI.https://go.nature.com/30xMnf1 -
With a growing aging population, research suggests a focus on reversing aging may be an economically more viable option than a focus on combating disease https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3938188/ …
#reverseaging#diseases#senescence#fitness#patternofhealthpic.twitter.com/rXhrq5D7ME
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