Rezultati pretraživanja
  1. ICYMI | In this News&Views, Katharina Wolter & Lars Zender explore how cancer therapies can trigger of tumour cells and a new study showing that eradication of therapy-induced senescent cells () can improve therapy

  2. 24. sij
  3. 24. sij

    How chronic turns your : Hyperactivation of sympathetic nerves drives depletion of melanocyte cells.

  4. REVIEW | Cellular in diseases: from pathogenesis to therapeutics

  5. 1. sij

    Great read to start the year. - CS-exposed p16-/- mice exhibit reduced emphysema, less ACECII senescence and better lung function.

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  6. 18. pro 2019.
  7. 21. lis 2019.
  8. 21. lis 2019.

    Two papers, published today in and , identify cardiac glycosides as a new class of senolytic drugs, active in vitro and in animal models. Work from Jesus Gil and

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  9. 4. ruj 2019.

    Des chercheurs américains sont parvenus à désactiver un des gènes responsables de la , cette pathologie qui accélère la . Ce n'est pas pour cela qu'on pourra ralentir le vieillissement !

  10. In a study supported by the Division of Cancer Biology, researchers et al. showed that the Kindlin2-p53-SerpinB2 signaling pathway regulates cellular in .

  11. 17. kol 2019.

    : hücrelerin sessiz moda geçmesi normal ve kemoterapi görmüş hücrelerde oldukça farklı seyrediyor. Normal hücre: p21 proteini “siklin bağımlı kinazlar”ı inhibe eder ve hücre bölünmez. Kemoterapi görmüş hücre: p21 hücre bölünmesini teşvik eder

  12. 2. kol 2019.

    Is signaling a driver or downstream effector of ? The p53-MDM2 axis is key to many aspects of both pathological and normal aging… read this beautiful review by Danyi Wu & Carol Prives on CDD !

  13. 30. srp 2019.

    “We're trying to study the reasons cells age, so that we might be able to design treatments for better aging." This researcher reflects on his investigation of :

  14. Our AUGUST issue out NOW has all of this: therapeutic vulnerabilities of paediatric solid tumours, , emerging roles of YAP & TAZ in the & alternative tumour-specific antigens for development

  15. 15. srp 2019.

    Knockout of Kindlin-2 via /Cas9 in several cell lines significantly increased expression levels of both SerpinB2 and p21 resulting in the activation of hallmarks of cellular . | In , by researchers from .

  16. With a growing aging population, research suggests a focus on reversing aging may be an economically more viable option than a focus on combating disease

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