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you ever ask "huh, i wonder why it's called this" and then immediately regret it as the absolute worst english etymology slams you in the facepic.twitter.com/WQ8QFMuGVi
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"An analysis of samples acquired four weeks after the German prohibition of JWH-018 took place found that the manufacturers had shortened the alkyl chain by one carbon to circumvent the ban" is an extremely cursed sentencepic.twitter.com/p4sREXeyA3
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just learned about efron's dice and my head hurts nowpic.twitter.com/2FPXi8ZtHf
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as usual, MCTB has an incredibly detailed and fantastic set of advice on what to do with "unwanted" emotions and thoughts. this is actually incredible so i'm just going to post the whole thing here.pic.twitter.com/Auz4dd8f4s
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on a related and interesting note, a thing i learned the other day is that there are some classes of drugs that are primarily metabolized by MAO in humans, rather than by P450, COMT, or other more familiar drug metabolism paths https://pdfs.semanticscholar.org/e50a/2c4c9477598c57290724f2e43ea9e623394d.pdf … http://sci-hub.tw/10.1111/j.1556-4029.2012.02289.x …pic.twitter.com/9ff0PIeFMw
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shulgin also has some great radioactive drug "stories" (h/t
@lukeweston)pic.twitter.com/1fll8afq1K
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and then!!! they confirmed that haematopoietic stem cells released from bone marrow are the actual progenitors of the fibrotic tissue, not proliferation in existing heart cellspic.twitter.com/tvQpkExq6a
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this incredibly odd investigation discovered that, at least in mice, BONE MARROW 5-HT2B RECEPTORS appear to be required for cardiac valvulopathy to happen. what. https://bpspubs.onlinelibrary.wiley.com/doi/pdf/10.1111/bph.13981 …pic.twitter.com/h1vNyPZcZs
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you may find this paper interesting, it's the best review i've found so far on the mechanisms of neurotoxicity https://www.karger.com/article/pdf/253548 …pic.twitter.com/01scLTJxU7
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new hobby: reading comments on papers instead of the papers themselves just to see the extremely dry snarkpic.twitter.com/O99v5d71fe
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found this interesting old-looking patient medical history form at my dentist’s that has “taken fen-phen” on itpic.twitter.com/X7QPyOlNEg
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hey, can any pharm-chem people explain to me how activation efficacy works? i had assumed it was a measure of agonism (e.g. 100 = full agonist), but 25I-NBOME is (iirc) a 5-HT2A full agonist, so this doesn't make sense.pic.twitter.com/G5VSQtgZHC
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seeing pharmacology papers citing Erowid gives me lifepic.twitter.com/vNXkXPhj6n
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TIL: norfenfluramine was actually a super-agonist for 5-HT2B. Jesus Christpic.twitter.com/w8IYDk0Tsc
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this story about how hallucinogens were found to be potent anti-inflammatories is absolutely wild https://erowid.org/culture/characters/nichols_david/nichols_david_interview1.shtml … (paper: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3788795/ …)pic.twitter.com/cDo7H6rThG
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by the way, speaking of neurotoxicity, there's some interesting hypotheses that a lot of studies tend to overestimate neurotoxicity because of the way their PET tracers work, as well as the more general problem of many studies not giving enough time to let receptors upregulatepic.twitter.com/x0dEzj53cS
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