That would be my starting point. Then I’d look at the risk of bias and how that might be impacting on the observed association. Study design here already sets my prior to “interesting, but....”
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^ this. Replication and (to a slightly lesser extent) pre-registration do nothing when the flaws are inherent to the study design.
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*laughcry*
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N = ~350, no main effect of genotype, GxE p-value of *just* under 0.05... I think we've been here before.
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Čini se da učitavanje traje već neko vrijeme.
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