A first case of #COVID19 reinfection from HKU, with distinct virus genome sequences in 1st and 2nd infection (142 days apart). Kudos to the scientists for this study.
This is no cause for alarm - this is a textbook example of how immunity should work.
(1/n)https://twitter.com/cwylilian/status/1297830744509698050 …
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2) Patient had no detectable antibody at the time of reinfection but developed detectable antibody after reinfection. This is encouraging. (3/n)pic.twitter.com/8OgylRxPyn
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3) Since reinfection can occur, herd immunity by natural infection is unlikely to eliminate
#SARSCoV2. The only safe and effective way to achieve herd immunity is through vaccination. (4/n)pic.twitter.com/tqtQOg8Fjf
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Lastly, while this is a good example of how primary infection can prevent disease from subsequent infection, more studies are needed to understand the range of outcomes from reinfection. (end)
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It will be interesting to see what childhood vaccinated viruses these C19 recovered patients become susceptible to later on due to the B cell damage done by C19. Would be a good time for some testing to see if this population needs a repeat of all childhood vaccinations.
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And it would be helpful to know how long any childhood vaccinations offer some protection, number of booster shots, etc.
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...."may have" protected, yes?
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Question: if this is an example of good immunity then what if the asymptomatic patients found during first wave were actually immune because SARS2 has been around longer? Or that similar common cold is enough to block disease but not infection?
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Can you explain how an infected person can be asymptomatic? AFAIK, viruses need to be actively infecting and destroying cells in order to replicate, and that should give some symptoms. They cannot survive for long by themselves.
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Suppressing the innate immune response . So shedding the virus but the immune system hasn't detected it as cognate antigen?
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