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  1. Prikvačeni tweet
    18. stu 2019.

    Our paper describing the role of T-type in homocysteinemia-induced mechanical eventually . A big 🙏 to our colleagues and for this collaborative work. ➡️ 1/5

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  2. 31. sij

    Very nice study from the Liddle lab showing that Piezo1-induced activation of leads to the development of ➡️

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  3. 23. sij

    ‼️ Beautiful work ‼️ β-adrenergic modulation of cardiac L-type elucidated! Is that the end of a 50-year-old debate? Beautiful work! ➡️

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  4. 14. sij

    KLHL1-dependent modulation of Cav3.2 T-type calcium expression controls mechanical 👍 From Piedras-Renteria lab ⁦

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  5. 13. sij

    De novo (disease causing?) mutation in Cav1.3 calcium associated with neurodevelopmental disorders ➡️

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  6. 20. pro 2019.

    The truth is complex - very nice paper documenting the detailed processing mechanisms of peripheral by lamina I spinal neurons ➡️

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  7. 18. pro 2019.

    Timely review article on the trafficking mechanisms of voltage-gated Nav1.6 ➡️

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  8. proslijedio/la je Tweet
    9. pro 2019.

    affects 30-50% of the world population. of in coll. with scientists from and demonstrated the causal implication of in the development of pain syndromes:

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  9. 26. stu 2019.
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  10. 25. stu 2019.

    Selective blockade of Nav1.7 by the toxin and analgesic properties. ➡️

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  11. 21. stu 2019.
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  12. 21. stu 2019.

    Congratulations to Leos for receiving 2nd best poster award mol biol panel at the student scientific conference 🍾🎉

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  13. 20. stu 2019.

    These guys have a campaign to complete a film on chronic - trailer ➡️

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  14. 20. stu 2019.
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  15. 19. stu 2019.

    Beautiful bioengineering work from the Colecraft lab describing a genetically-encoded blocker of voltage-gated

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  16. 18. stu 2019.

    Finally, we identified PKC-dependent phosphorylation sites requeiered for homocysteine-dependent modulation of Cav3.2 channels. 5/5

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  17. 18. stu 2019.

    In vitro, we show that homocysteine enhances recombinant T-type calcium currents by promoting the recycling of Cav3.2 channels back to the plasma membrane through a protein kinase C-dependent signaling pathway. 4/5

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  18. 18. stu 2019.

    Homocysteinemia-induced mechanical allodynia was reversed on pharmacological inhibition of T-type calcium channels. 3/5

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  19. 18. stu 2019.

    We used a rodent model of experimental homocysteinemia to demonstrate the causal association between homocysteine and the development of mechanical allodynia. 2/5

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  20. 18. stu 2019.

    Tidy study from Edward Tate lab using toxin-based photocrosslinking probes identified new selective binding sites within .7 providing important clues for the development of therapeutics ➡️

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