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Prikvačeni tweet
Our paper describing the role of T-type
#calcium#ionchannels in homocysteinemia-induced mechanical#pain eventually#openaccess@PAINthejournal. A big
to our colleagues @HotchkissBrain and@KazanUni for this collaborative work.@IOCBPrague
https://bit.ly/2KtaohO 1/5pic.twitter.com/VNd6O3xGaD
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Very nice study
@jclinicalinvest from the Liddle lab@dukemedicine showing that Piezo1-induced activation of#TRPV4#ionchannels leads to the development of#pancreatisis
https://bit.ly/2RIE4eP pic.twitter.com/MqakosaRbj
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Beautiful work
β-adrenergic modulation of cardiac L-type #calcium#ionchannels elucidated! Is that the end of a 50-year-old debate? Beautiful work!
https://go.nature.com/2NOI8rt pic.twitter.com/9YFzgBaBuy
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KLHL1-dependent modulation of Cav3.2 T-type calcium
#ionchannels expression controls mechanical#pain
From Piedras-Renteria lab @LoyolaChicagohttps://www.frontiersin.org/articles/10.3389/fnmol.2019.00315/full …Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
De novo (disease causing?) mutation in Cav1.3 calcium
#ionchannels associated with neurodevelopmental disorders#channelopathies#autism
https://bit.ly/30gpSfL pic.twitter.com/h74Xm6hZWX
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The truth is complex - very nice paper documenting the detailed processing mechanisms of peripheral
#nociception by lamina I spinal neurons#ionchannels#pain
https://go.nature.com/2PIPhuZ pic.twitter.com/94IsDE8Al3
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Timely review article on the trafficking mechanisms of voltage-gated
#sodium#ionchannels Nav1.6#channelopathies
https://bit.ly/2rXdODm pic.twitter.com/WgpmSfbR2H
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Norbert Weiss proslijedio/la je Tweet
#chronicpain affects 30-50% of the world population.@TheWeissLab of#IOCBPrague in coll. with scientists from@UCalgary and@KazanUni demonstrated the causal implication of#homocysteinemia in the development of pain syndromes:https://www.uochb.cz/en/news/144/chronic-pain-driven-by-metabolic-dysfunction …Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
First structure of a T-type
#calcium#ionchannel at atomic resolution!
https://twitter.com/osamaharraz/status/1199361417826258950 …Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Selective blockade of Nav1.7
#sodium#ionchannels by the#HpTx3 toxin and analgesic properties.#pain
https://www.mdpi.com/2072-6651/11/12/680 …pic.twitter.com/5sWwE48XWz
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#postdoc position in Sam Young lab (@SamYoungLab)@UIowaNeuro, Iowa City
- calcium #ionchannels in neuronal circuits
https://young.lab.uiowa.edu/job-opening #scijob#scicareerhttps://twitter.com/SamYoungLab/status/1197554820720287747 …
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Congratulations to Leos
@TheWeissLab@IOCBPrague for receiving 2nd best poster award mol biol panel at the student scientific conference@VSCHT
pic.twitter.com/B34463AC9b
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These guys have a
#crowdfunding campaign to complete a#documentary film on chronic#pain -#PainBrain trailer
https://vimeo.com/370252628 https://painbrainfilm.com/ Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Exciting
#PhD program@imed_tweets, Innsbruck
to work on several aspects of #calcium#ionchannels. Call open until December 2, 2019!#sciencecareers#sciencejobs
https://phd-school.i-med.ac.at/events/nbsp/?tx_news_pi1%5Bnews%5D=344&cHash=d5f8dddfb2d39376ed9d2b130252826d …https://twitter.com/jerriska_gerald/status/1197103151662411776 …Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Beautiful bioengineering work from the Colecraft lab describing a genetically-encoded blocker of voltage-gated
#calcium#ionchannels @eLifehttps://elifesciences.org/articles/49253Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Finally, we identified PKC-dependent phosphorylation sites requeiered for homocysteine-dependent modulation of Cav3.2 channels. 5/5
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In vitro, we show that homocysteine enhances recombinant T-type calcium currents by promoting the recycling of Cav3.2 channels back to the plasma membrane through a protein kinase C-dependent signaling pathway. 4/5
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Homocysteinemia-induced mechanical allodynia was reversed on pharmacological inhibition of T-type calcium channels. 3/5
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We used a rodent model of experimental homocysteinemia to demonstrate the causal association between homocysteine and the development of mechanical allodynia. 2/5
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Tidy study from Edward Tate lab
@TateScience using toxin-based photocrosslinking probes identified new selective binding sites within#Nav1.7#ionchannels providing important clues for the development of#pain therapeutics
https://www.sciencedirect.com/science/article/pii/S2451945619303563 …pic.twitter.com/tugxkk6bkl
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