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Thomas Gatliff proslijedio/la je Tweet
Loss of NAD during aging inhibits nuclear-encoded genes for mitochondrial proteins. New work suggests it inhibits DNA repair, releasing pieces of nuclear DNA into the cytoplasm, causing SASP inflammation in aging & cancer https://tinyurl.com/ws8glv6 https://tinyurl.com/ttdvcsw
#Cancerpic.twitter.com/gM4dwx5dTK
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Thomas Gatliff proslijedio/la je Tweet
Senescent cells display cytoplasmic chromatin that activates cGAS-STING and inflammation. But what triggers cytoplasmic chromatin? Mitochondria play a role. From Peter Adams lab
@PeterAdamsLab and our lab. Led by@MG_Vizioli http://m.genesdev.cshlp.org/content/early/2020/01/28/gad.331272.119 …Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Thomas Gatliff proslijedio/la je Tweet
A high‐fat diet reverses metabolic disorders and premature aging by modulating insulin and IGF1 signaling in SIRT6 knockout micehttps://onlinelibrary.wiley.com/doi/full/10.1111/acel.13104 …
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Thomas Gatliff proslijedio/la je Tweet
Calorie restriction-mediated lifespan extension is independent of Nrf2 in micehttps://www.sciencedirect.com/science/article/abs/pii/S0891584919323652 …
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Thomas Gatliff proslijedio/la je Tweet
The list they came up with. Hopefully it will inspire further research along the same lines.pic.twitter.com/U5uxtvA8G7
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Thomas Gatliff proslijedio/la je Tweet
Final version of methods paper on workflow for quantifying calcium/bioelectricity tracking in frog embryos, by Patrick McMillen (from our lab) and Richard Novak, a great collaborator and scientist at
@wyssinstitute :https://authors.elsevier.com/a/1aU2954HFDeiEHvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Thomas Gatliff proslijedio/la je Tweet
Unlike more robust species, we mammals lose our hearing because we fail to regenerate damaged cochlear hair cells. New study shows long-term activation of Sirt1 with resveratrol increases mitochondria & reduces hair cell loss in old BL6 mice. Music to our
https://tinyurl.com/wqlvga6 pic.twitter.com/i3Zou4cmbg
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Thomas Gatliff proslijedio/la je Tweet
High insulin levels (like with
#insulinresistance) activate inflammation and inhibit autopaghy in joint cells; may be a mechanism wherein IR links to#arthritis. https://bit.ly/2RQVvJ3 pic.twitter.com/MLnP8J0q2U
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Thomas Gatliff proslijedio/la je Tweet
Tissue specificity of
#senescent cell accumulation during physiologic and accelerated aging of micehttps://onlinelibrary.wiley.com/doi/full/10.1111/acel.13094 …Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Thomas Gatliff proslijedio/la je Tweet
Hyperactivation of TORC1 Drives Resistance to the Pan-HER Tyrosine Kinase Inhibitor Neratinib in HER2-Mutant Cancers:https://www.cell.com/cancer-cell/fulltext/S1535-6108(19)30583-5?dgcid=raven_jbs_aip_email …
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Thomas Gatliff proslijedio/la je Tweet
Transplanting cells from old but not young donors causes physical dysfunction in older recipientshttps://onlinelibrary.wiley.com/doi/10.1111/acel.13106#.XimuDFdhoWY.twitter …
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Thomas Gatliff proslijedio/la je Tweet
The Future is Now! In Vivo AAV-CRISPR/Cas9–Mediated Gene Editing Ameliorates Atherosclerosis in Familial Hypercholesterolemia https://doi.org/10.1161/CIRCULATIONAHA.119.042476 …
@skathire@pabeda1@SABOURETCardio@ProfKausikRay@rauldsf_santos@KarleziAlonso@Drlipid@lipiddoc@MeralKayikciog1pic.twitter.com/YoQjUKkbXv
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Thomas Gatliff proslijedio/la je Tweet
mTORC1 directly inhibits AMPK. Well it would, wouldn’t it. Discovery made in yeast by the wonderful Janni Petersen at Flinders Uni, my team confirmed in mammals. A simple story with 157 Westerns at the last count
https://twitter.com/NatMetabolism/status/1219340839647760387 …
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Thomas Gatliff proslijedio/la je Tweet
Anyone interested in anti-aging / life extension should give a follow to
@BlagosklonnyHvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Thomas Gatliff proslijedio/la je Tweet
The
#mTOR hypothesis of aging is still alive but our work and others show that we need to find better ways of selectively inhibiting mTORC1, and avoid inhibiting mTORC2 or other kinases that are necessary for health and survivalHvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Thomas Gatliff proslijedio/la je Tweet
Collaboration with
@WaskoBM on the link between vacuole/lysozomes, mitochondria, and aging in individual cells is starting clarify different trajectories of aging.https://www.biorxiv.org/content/10.1101/2020.01.05.895433v1 …Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Thomas Gatliff proslijedio/la je Tweet
Complete removal of food (all adult life) prolongs life of the worm C elegans. So all adult life the aging worm is doing something that activates mTOR and shortens its life. Why? Aging is mTOR-driven quasi-program, a continuation of developmental growthhttp://shorturl.at/kqD79
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Thomas Gatliff proslijedio/la je Tweet
Probably best medical resources in the world. Yet, our country has the lowest longevity of developed countries &
. Also, highest expenditure by far..But the epidemic of cardiometabolic disease, Alzheimer’s and (maybe?) cancer hasn’t started yet...Economic collapse inevitable.pic.twitter.com/WRuZtIXs0g
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Thomas Gatliff proslijedio/la je Tweet
Couldn’t agree more!!! Yes!!! You are right as always!!! Please read and re-read this editorial (2010). Every word is worth it. Am J Cardiol 2010;106:1364–1366pic.twitter.com/49U5uCCUvv
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Thomas Gatliff proslijedio/la je Tweet
One of the most important science debates of the decade. Gary Taubes versus Stephan Guyenet, March 19th, on the Joe Rogan Experience. Why this is so important.https://is.gd/WQgR0y
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