Okay, it's time. Heritability in Five Easy Tweets: Imagine studying a cohort of individuals (of the same species, let's say human) in a particular set of environmental circumstances. You assess everyone for some observable trait & find it varies from one person to the next...1/5
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Of course, there are wonderful systematic errors in self-reports that are, well, heritable in twin studies....
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GCTA only accounts for Additive (linear) heritability; mid-level personality constructs (Big 5) consistently include Dominance (non-linear) heritability, even with quality measures (longer, not just self-report; more common in twin studies)https://link.springer.com/article/10.1007/s10519-014-9654-x …
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Level of construct may be part of the issue. Dominance goes away when looking at Personality as a combo of general & facet-level influences (in prep, ppt from June 2017 with twins in MIDUS + GWAS data in HRS):https://osf.io/awrj8/
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For number of constructs etc., I had in mind stuff like https://www.sciencedirect.com/science/article/pii/S019188691400347X … and https://www.personality-project.org/revelle/presentations/brazil.pdf … No clean structure in personality at any number of factors or levels etc. Big mess.
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But no different than clinical (which
@drderringer can inform us about too) where they've done some nice hierarchical twin work (thinking Bob Krueger). I don't think we have a clear understanding of what level is ideal for a genetic test (broad or narrow). > -
If the work on endophenotypes is any indication, getting more specific and narrow does not lead to clarity. Nor do higher order factors (the Big 2) render in genetic space any better than the Big Five. >
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I think the measurement in personality is as good (or bad) as clinical psych and therefore the difference is not a result of psychometrics, but something else. I suspect development is the culprit, but I'm biased toward thinking in those terms.
End of conversation
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