Chirag Parikh

@KidneydrChirag

nephrologist, translational researcher, dad

Vrijeme pridruživanja: studeni 2014.

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  1. 3. velj

    In this editorial, we discuss management of creatinine elevations (??AKI) with intensive BP regimen to achieve SPRINT goals. Majority of creatinine rise not associated with injury- it is permissive hypercreatininemia. Algorithm below. Let us know your thoughts.

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    Odgovor korisniku/ci
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    8. sij
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  4. 8. sij

    Huge Variation in recovery and transplant of deceased donor AKI kidneys....

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  5. 8. sij

    In the propensity matched analysis of deceased donors, kidneys with AKI have similar outcomes as non-AKI kidneys. Also, large variation across transplant centers utilizing AKI kidneys. Great opportunity to expand deceased donor pool with AKI kidneys.

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  6. 26. pro 2019.

    Enjoyed developing this editorial with . Lots of unanswered questions and opportunities to optimize RAAS inhibition in the setting of AKI- we believe majority of the bumps in creatinine are not intrinsic kidney injury.

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    Nice work ! Renin-Angiotension System Blockade after Acute Kidney Injury

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  8. 13. stu 2019.

    Congratulations! Although only 10 years old, the consortium has uplifted the landscape of kidney disease knowledge with its immense contributions.

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  9. 10. stu 2019.

    Congratulations, to for this wonderful achievement and for many years of hard work to enhance education and knowledge on kidney diseases.

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    Newly established ‘mid-career’ awards announcing the awards @dwein003 Tamara Isikova of and more Congrats to all the awardees!

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    6. stu 2019.

    Academic Nephrology Opportunity at Johns Hopkins/Baltimore job with Johns Hopkins University School of Medicine, Div of Nephrology Baltimore, Maryland

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  12. 2. stu 2019.

    If donor quality had a huge role in recipient outcomes, the kidney pairs from same donor in two recipients should have correlated outcomes. We saw only minimal concordance in donor pairs after rigorous testing. Can we justify discarding >3000 kidneys/year on donor quality?

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  13. 2. stu 2019.

    Fenofibrates joins other agents such as ACE/ARBs, SGLT2 inhibitors, etc where biomarkers help clarify that bumps in creatinine are not due to injury to the kidney.

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  14. 21. lis 2019.

    Enjoyed developing the editorial with . We hope more patients can get cardioprotective medications. “Permissive AKI” with treatment of heart failure

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  15. 11. lis 2019.

    Thank you for inviting me. Had a great time meeting everyone at UVA

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  16. 30. ruj 2019.

    Clinical trials in contrast nephropathy require 6000-10,000 patients as outcomes of doubling, dialysis and death are rare. We show that biomarkers of injury can enrich the event rates and reduce the trial size by 30-40%! Huge cost savings!

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  17. 24. ruj 2019.

    ⁩ and the ⁦ investigators finalizing the AKI adjudication protocol before the first AKI biopsy

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  18. 12. ruj 2019.

    Yes-the challenge is to determine the Rolle’s point on the curve- the inflection point-when compensation ends and decompensation starts (in medicine) or more taxation will reduce revenue in economy. There is a Rolle’s point for every organ and every economy!

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  19. 10. ruj 2019.

    As classification, diagnostic criteria and pathogenic theories have evolved since the traditional description of HRS-1 and HRS-2, we have summarized novel aspects and new classification framework for hepatorenal syndrome in this position paper.

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  20. 10. ruj 2019.

    By linking 7 datasets, 10 year longitudinal follow-up of 3600 children after surgical repair for CHD demonstrates good progress but we can do better to reduce incidence of ESKD and mortality. Maybe better control of hypertension?

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