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VERY nice! Glad to see models seem to be able to capture observation, now we just need to figure out the (super weird...) biology behind 
.
Question - latency up to beginning of outbreak makes sense, but that hypothesis can be directly tested (given data). I.e., what's the clock within each of the two DRC outbreaks? I assume probably the same as other outbreaks? (i.e., close to larger tree).
If yes, then we know that latency is a function of the (reservoir?) host, and not actually something intrinsic to a particular (DRC) Ebola virus lineage. Completely what we would expect, but nice to explicitly test (especially given, ehm, certain papers...).
I think it may be a mechanism that the virus has used to persist in the reservoir - sweeps through a population (roost?) and if it isn’t able to move on to another, then much later it can re-emerge from an older bat to infect all of the new naive bats that have been accumulating.
The point of the post is that the best fit is that there was a period of latency back in shared lineages (and then became non-latent). It has to be replicating to produce new infections (either in the non-human or human).
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