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Excited to have a new outstanding cancer immunologist
@PaulosLab to work with@WinshipAtEmory!https://twitter.com/PaulosLab/status/1223997187639664647 …Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Great writeup of our recent work by
@UlrikeHarjes from@NatureRevCancer. Always nice when other people do a better job explaining your work than you!https://twitter.com/NatureRevCancer/status/1218174886763474944 …
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Haydn Kissick, PhD proslijedio/la je Tweet
Letter from
@profshanecrotty & co on "Reinvigorating NIH Grant Peer Review" | Encourages immunologists & colleagues throughout biomedical research funded by the NIH to take the “Once-a-Year” pledge & provides suggestions to improve grant study sections https://authors.elsevier.com/a/1aP1-3qNrUfz2D …pic.twitter.com/bk06prsbPu
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Notice the small piles of chillies on the side if everyones plate. Spiiiiicy!!!https://twitter.com/KateJeffrey1/status/1207508017958313986 …
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When the pre-activated T-cells find it, they can receive all the signals they need to become killers. Could be a 2-step process to stop over production of killers when only a localized response is needed. Still a lot to understand about this obviously.
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When T-cells encounter these conditions, they might only activate enough to start scanning for regions of increased inflammatory signals such as these outposts in the tumor. This outpost is then a way to concentrate the limiting amounts of danger signals and antigen.
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then travel to this structure to encounter antigen again before taking the final step of its differentiation. Seems like a bad design. My wild guess is that there is a lack of ‘danger signals’ or low concentrations of tumor antigen in the peripheral sites.
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There is a perfectly good lymph node with dendritic cells joined directly to the cancer by blood/lymphatic vessels. Why go to the trouble of building an outpost of these cells in the tumor. It seems to me a T-cell would need to see antigen in a lymph-node,
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Lots of questions to follow up on. Mechanisms that cause the generation, how tumors might inhibit to escape T-cell destruction, if these structures correlate with response to checkpoint therapy etc. etc. What I think is most interesting is why the immune system does this.https://twitter.com/nature/status/1205628471109672960 …
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Thanks to all our friends and collaborators over the years who helped us with this work. This work was only possible
@EmoryUniversity with our outstanding physician and immunologist collaborators!https://twitter.com/careyjans/status/1204827552151474176 …Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Check out the first article from our lab in
@NatureComms! Describing how novel non-coding RNAs undergo changes during T cell differentiation. https://www.nature.com/articles/s41467-018-07956-7 … Thousands of previously unannotated genes, many shared in human and mouse. Great work Will Hudson &@nataliya_pro_Hvala. Twitter će to iskoristiti za poboljšanje vaše vremenske crte. PoništiPoništi -
Thanks for all the kind welcomes, very excited to join all you friendly tweeters!
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