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GidMK's profile
Health Nerd
Health Nerd
Health Nerd
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@GidMK

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Health NerdVerified account

@GidMK

Epidemiologist. Writer (Guardian, Observer etc). "Well known research trouble-maker". PhDing at @UoW Host of @senscipod Email gidmk.healthnerd@gmail.com he/him

Sydney, New South Wales
theguardian.com/profile/gideon…
Joined November 2015

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    1. Health Nerd‏Verified account @GidMK May 29

      A "new study" has hit the headlines that apparently proves that SARS-CoV-2 was lab-grown I am QUITE SKEPTICAL for a number of reasons, would love your thoughts...pic.twitter.com/J9aJc1BeTq

      21 replies 49 retweets 184 likes
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    2. Health Nerd‏Verified account @GidMK May 29

      First massive red flag🚩: the paper is NOT PUBLISHED YET Science journalists know you NEVER report on a paper that is unpublished (because it might be shit)

      4 replies 5 retweets 104 likes
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    3. Health Nerd‏Verified account @GidMK May 29

      Second 🚩🚩🚩: the abstract is...kind of weird I'm not a virologist, but talking about some previous experiments that "reverse the burden of proof" is...not really logicalpic.twitter.com/9GUPW0Bt3Q

      5 replies 3 retweets 63 likes
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    4. Health Nerd‏Verified account @GidMK May 29

      Also, the statement in the abstract that "all relevant" information has been destroyed or is unavailable is both untrue and a weeeird conspiracy theory for a scientific journal to publish

      3 replies 1 retweet 65 likes
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    5. Health Nerd‏Verified account @GidMK May 29

      Reading the abstract, they literally say "because of our study, which does not prove anything, we expect other people to prove that the virus did not escape from a lab" Like I said, not ideal logic that

      3 replies 0 retweets 71 likes
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      Health Nerd‏Verified account @GidMK May 29

      In an interview, one of the two scientists also says things that are, uh, wrong Again, not a virologist but as I understand it this is simply untrue (natural viruses can do lots of things)pic.twitter.com/G9G3KKvEnB

      5:44 PM - 29 May 2021
      • 1 Retweet
      • 67 Likes
      • Mathematiker plädiert für Ruhe und Rationalität Taylor Nichols, MD ☀️🦅Daya G.✨🐢 Jonathan Cronk Amir Laura Henderson Mark Boyle Kyle Swanson MD
      10 replies 1 retweet 67 likes
        1. Health Nerd‏Verified account @GidMK May 29

          Anyway, would love some virologists perspectives on this because it does not seem likely despite being everywhere in the news @MackayIM @angie_rasmussen @trvrb https://www.dailymail.co.uk/news/article-9629563/Chinese-scientists-created-COVID-19-lab-tried-cover-tracks-new-study-claims.html …pic.twitter.com/shMz0HKj0Q

          32 replies 1 retweet 56 likes
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        2. Dr KayRay‏ @ktibus May 29
          Replying to @GidMK

          From an evolutionary biology perspective this is an extremely simplistic view.

          1 reply 0 retweets 1 like
        3. Well-Sapiens (Ralph Einfeldt) 🏊‍♂️ 🚴‍♂️ 🏃‏ @SapiensWell May 30
          Replying to @ktibus @GidMK

          It‘s not simplistic ist is just plain wrong. Only if a Virus gets so deadly that it limits its spread there will be selection advantage for the less deadly variants.

          0 replies 0 retweets 0 likes
        4. End of conversation
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        2. Marianne - TiredDoc 💉 🇫🇷 🇬🇧 🇦🇺‏ @Amalgamquietude May 29
          Replying to @GidMK

          You’re right, that’s definitely not true, see “the Spanish flu pandemic”. Also, how can you manufacture a virus in such a way that you can then predict its future behaviour once it’s loose? Also not a virologist unfortunately, but will be interesting to see other opinions.

          1 reply 0 retweets 6 likes
        3. Sanford May‏ @sanfordmay May 29
          Replying to @Amalgamquietude @GidMK

          I’ll second that “tell that to the Spanish flu”

          0 replies 0 retweets 3 likes
        4. End of conversation
        1. Timlagor‏ @Timlagor May 29
          Replying to @GidMK

          This is pure bullshit: there's a *reason* virus variants that are less lethal generally have a replication advantage and it doesn't apply (strongly) to Covid-19 because Covid gets most of its infection spread done in the pre-symptomatic-and early symptomatic period.

          0 replies 0 retweets 1 like
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        2. Nicholas Nelson‏ @yesitsnicholas May 29
          Replying to @GidMK

          We can expect this of highly fatal diseases that mostly spread when nearing host death - if the host is dying too fast to spread (e.g. Ebola), less fatal / less rapidly fatal variants may have selective advantage. COVID19 spreads well during asymptomatic & nonfatal infection. 1/2

          1 reply 0 retweets 0 likes
        3. Nicholas Nelson‏ @yesitsnicholas May 29
          Replying to @yesitsnicholas @GidMK

          So, natural selection: the huge delay between transmissible time - when many COVID-positive people are still physically well and out in the world - and eventual death from COVID, means minimal selective pressure AGAINST its morbidity but still pressure FOR transmissibility. 2/2

          1 reply 0 retweets 0 likes
        4. Show replies
        1. HT‏ @HansiiT May 29
          Replying to @GidMK

          It basically happened with B.1.1.7. We are one year in knowing that virus, still arguing what the death rate of B1 is. So it is quite a whombat-shit statement.

          0 replies 0 retweets 0 likes
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        2. snarkydog‏ @snarkydog2 May 29
          Replying to @GidMK

          This is, um, really dumb. Viruses are parasites. Parasites compete with each other for access to hosts. If hosts are plentiful, then rapid replication within hosts is the best strategy. If hosts aren't plentiful, then slow replication is better.

          1 reply 0 retweets 0 likes
        3. snarkydog‏ @snarkydog2 May 29
          Replying to @snarkydog2 @GidMK

          And, guess what. High replication rate=high virulence (because, of course, more host resources are being used up). Slow replication=low virulence. So, evolution of virulence depends on access to hosts, and competition from other parasites (viruses here).

          0 replies 0 retweets 0 likes
        4. End of conversation

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