The continued weird flex from the keto community to bash statins/LDL is unhelpful. a low CHO, high fiber from LC veggie, high MUFA w/ some mixed PUFA fat diet pretty much crushes every single marker of metabolic dz we have yet theres continued resistance becuz antiestablishmenthttps://twitter.com/KCKlatt/status/1212848489207603202 …
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For something like atherosclerosis that takes decades to develop an ARR 1% is fantastic.
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Not fantastic for the 99% users who would not reap a benefit. Longer life with LDL>100. What if statin benefit from its
NO or from its NF-KappaB inhibition?
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I hope I miss read this
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The CETP inhibitors failed miserably— https://www.google.com/amp/s/www.nytimes.com/2016/04/04/health/dashing-hopes-study-shows-cholesterol-drug-has-no-benefits.amp.html …. One PCSK9 inhibitor trial had more all-cause death with drug group
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Just to make sure I understand: If my current 10y risk for MACE is 10%, and therefore according to current guidelines calls for the prescription of statins, the use of statins will reduce my risk to 9%? If so, why is this impressive (for me)? What an I missing?
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