How are the results in epilepsy not real effects?
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Replying to @KetoCarnivore @AlastairMcA30
They are. But you wish to extrapolate this to different conditions, something which I do not believe there is evidence for
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Replying to @GidMK @AlastairMcA30
I didn't say anything about that. I said that MAs using a threshold of 40% are inherently unable to find appropriate results.
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Replying to @KetoCarnivore @AlastairMcA30
Right. And I'm arguing that you are basing that off a theory, rather than fact
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Existence of ketones on LC is well established measurable physiological fact that you will find in any textbook and is measurable! They are only produced when insulin and glucose is low. You are arguing from position of ignorance
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Indeed. Ketosis is not theoretical, not are the metabolic downstream effects on countless things like lipids, hormones, autophagy, HDAC inhibition, etc. Just because the results in therapeutic contexts are less well studied doesn't mean the physiological state is in question.
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? I did not argue about ketosis. You have specified a threshold effect for low-carb above which we would see no benefit. My point is that this is not demonstrable and does not appear to be supported in the literature
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Maybe we're misunderstanding each other. I've been trying to say low carb diets have a specific physiological effect but only below a certain threshold <<<40%, that this is what low carb researchers mean by low carb, and that setting at 40 will inherently dilute findings.
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Replying to @KetoCarnivore
Right, and I'm saying that there may be a physiological impact, but in terms of benefits there is no evidence for a threshold effect and saying so is mostly theoretical rather than demonstrable
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Oh absolutely. But inferring across disease states has some obvious issues
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