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GidMK's profile
Health Nerd
Health Nerd
Health Nerd
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@GidMK

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Health NerdVerified account

@GidMK

Epidemiologist. Writer (Guardian, Observer etc). "Well known research trouble-maker". PhDing at @UoW Host of @senscipod Email gidmk.healthnerd@gmail.com he/him

Sydney, New South Wales
theguardian.com/profile/gideon…
Joined November 2015

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    1. L. Amber O'Hearn‏ @KetoCarnivore 22 Aug 2018
      Replying to @GidMK @bokkiedog and

      Ok. It would help me a lot as a data scientist, to understand why you think it's misapplied. I'm sincerely happy to be corrected on this point, although admittedly I'd be happier to be correct!

      1 reply 0 retweets 2 likes
    2. Health Nerd‏Verified account @GidMK 22 Aug 2018
      Replying to @KetoCarnivore @bokkiedog and

      I'd argue that the causal pathway is far less direct that you are implying, and that there diabetes as a proxy for a mediating variable makes little sense anyway. If anything, it would be a direct mediator of the causal relationship, rather than a tangential variable

      1 reply 0 retweets 0 likes
    3. L. Amber O'Hearn‏ @KetoCarnivore 22 Aug 2018
      Replying to @GidMK @bokkiedog and

      Thank you. I think I see the source of the conflict. Response pending; it's family time.

      1 reply 0 retweets 3 likes
    4. Health Nerd‏Verified account @GidMK 22 Aug 2018
      Replying to @KetoCarnivore @bokkiedog and

      Health Nerd Retweeted Health Nerd

      No worries. Family time is much more important than Twitter arguments If you want to know the criticism I'd personally make of the study, I did a quick thread herehttps://twitter.com/GidMK/status/1032390899995557888 …

      Health Nerd added,

      Health NerdVerified account @GidMK
      You could, for example, point out that the absolute risk was minimal and that the practical implications have been wildly overblown by most of the people reporting on the study
      Show this thread
      1 reply 0 retweets 1 like
    5. L. Amber O'Hearn‏ @KetoCarnivore 23 Aug 2018
      Replying to @GidMK @bokkiedog and

      I see two related reasons for the outcry against this study from low carb advocates (hereafter LCAs). First, the authors have implicated "low carbohydrate diets", when they have not studied any carb intake remotely like low carb diets (LCD). This matters, because 1/

      1 reply 0 retweets 2 likes
    6. L. Amber O'Hearn‏ @KetoCarnivore 23 Aug 2018
      Replying to @KetoCarnivore @GidMK and

      not only is there a huge gap between their lowest quartile at 38% and the <5% that LCDs entail, but the difference is critical physiologically. Only at those very low levels are symptoms of MetS demonstrably reversed, including the dyslipidemia of triglycerides and HDL. 2/

      1 reply 0 retweets 2 likes
    7. L. Amber O'Hearn‏ @KetoCarnivore 23 Aug 2018
      Replying to @KetoCarnivore @GidMK and

      This framing has caused harm to public perception of LCDs, exacerbated by inappropriate but predictable headlines such as "LCDs take four years off your life." It is difficult not to attribute deliberate deceptive intent, and this generates ill will and lack of trust. 3/

      1 reply 0 retweets 2 likes
    8. L. Amber O'Hearn‏ @KetoCarnivore 23 Aug 2018
      Replying to @KetoCarnivore @GidMK and

      The deeper conflict is what we were discussing. It is precisely that we don't agree on the causal pathways in the first place, that makes LCAs see this kind of study as fundamentally worthless. These assumptions affect how the statistics are analysed. 4/

      1 reply 0 retweets 2 likes
    9. L. Amber O'Hearn‏ @KetoCarnivore 23 Aug 2018
      Replying to @KetoCarnivore @GidMK and

      LCAs came to be advocates because of effects like the ones cited above. Even if you don't take carbohydrates as a primary driver of metabolic syndrome, once MetS has developed, risk factors can be drastically, favourably changed by LCDs. 5/

      1 reply 0 retweets 2 likes
    10. L. Amber O'Hearn‏ @KetoCarnivore 23 Aug 2018
      Replying to @KetoCarnivore @GidMK and

      The LCA model of causality is that (chronic high carb intake & some other factors) → chronic hyperinsulinemia → (MetS & diabetes & CVD), whereas the HCA model seems to be: (gluttony & sloth & genetics & some other factors) → overweight → (optionally MetS & diabetes) → CVD 6/

      2 replies 0 retweets 2 likes
      Health Nerd‏Verified account @GidMK 23 Aug 2018
      Replying to @KetoCarnivore @bokkiedog and

      Just saw this. It's an odd argument, because it is trivial to disprove the idea that high carb intake is a necessary precursor to hyperinsulinemia. The DIReCT trial, for example, had people reverse diabetes eating almost 100% carbs

      3:20 PM - 23 Aug 2018
      1 reply 0 retweets 0 likes
        1. L. Amber O'Hearn‏ @KetoCarnivore 23 Aug 2018
          Replying to @GidMK @bokkiedog and

          Sorry for the ambiguity. The arrow doesn't mean "necessary", and with the "other factors" not sufficient, either. As to the results of essentially all carb diets, I've read about Walter Kempner and others via @deniseminger https://deniseminger.com/2015/10/06/in-defense-of-low-fat-a-call-for-some-evolution-of-thought-part-1/ … I think it's important.

          0 replies 0 retweets 0 likes
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