Buck Institute

@BuckInstitute

Our mission is to end the threat of age-related disease for this and future generations

Novato, CA
Vrijeme pridruživanja: studeni 2010.

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  1. prije 12 sati

    Buck CEO gives a great overview of the goals and focus of at the Buck. And shares some of his best advice to , too!

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  2. prije 15 sati

    Stopping growth and proliferation can be just as important as starting. New information from shows the gene mob4 to be an important suppressor of tissue growth by downregulating in stem cells.

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  3. 3. velj

    Oh Mylanta! New details on why lysosomes need to be acidic—it’s essential to recycle iron into its nutrient form.

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  4. 2. velj
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  5. 1. velj

    Why does make hair gray? Looks like a flood of norepinephrine from sympathetic nerves causes pigment cells to overactivate and deplete their reserve. So technically does stress make your hair darker before it goes gray?

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  6. 31. sij

    In vacuole-impaired yeast, cysteine buildup disrupts iron homeostatis, is toxic to the mitochondria, and facilitates age-related mitochondrial decline. Iron supplementation reverses the damage!

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  7. 30. sij

    Just published in ! Astrocyte promotes glutamate toxicity in cortical neurons, filling in some gaps in our understanding of senescence and . Congrats to all the Buck authors!

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  8. 29. sij

    Join the Buck! The Buck AI Platform (BAIP) in collaboration with the Global Consortium for Reproductive Longevity and Equality at the Buck has an opening for a Machine Learning Data Scientist or Bioinformatics fellow. For more info:

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  9. 28. sij

    Sunlight may be required for normal energy metabolism—fat cells respond to sunlight and don’t burn as well without it, increasing the risk for metabolic syndrome. Just what we (don’t) want to hear in these dark, hungry days of winter…

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  10. 27. sij

    Join the Buck team! The Schilling lab is seeking a Postdoctoral Fellow to participate in innovative research aimed at understanding the biological and chemical processes that are common to both age-related diseases and aging.

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  11. 27. sij
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  12. proslijedio/la je Tweet
    27. sij

    Our understanding of the biology of has progressed to a point where viable therapeutics are entering early clinical trials. But it's a field that's still clouded with hype & scepticism. Download the Trends report

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  13. 26. sij

    One way to ? Reduce air pollution. A new study using and 40 billion observations found pollution events impact mortality for older adults. And improved air quality can save $$$ in medical costs!

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  14. 25. sij

    In data from a new study, women with premature menopause (before 40) were twice as likely to develop >1 chronic disease by the age of 60, and three times as likely to develop >1 chronic disease from the age of 60 onwards. Lets fix it!

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  15. 25. sij

    Is smoother skin just a good night’s sleep away? maintenance and repair respond to a rhythm and are disrupted when the body clock genes are disrupted (in mice)

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  16. 23. sij

    Join the Buck team! The Melov lab is seeking a Postdoc Fellow for innovative research aimed at understanding role of cellular senescence in Alzheimer’s disease, l focusing on single cell genomics, including induced pluripotent stem cells (iPSCs).

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  17. 22. sij

    More details on research that shows 500% lifespan extension in C. elegans: Researchers gave the worms the huge leap in lifespan when they engineered mutations in two different cellular pathways known to influence aging and lifespan.

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  18. 21. sij

    Boosting in aging flies ⬆ and restores heart function almost completely. Flies with overexpressed mTORC2 also had longer

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  19. 20. sij

    "Mitochondrial failure is a feature of many neurodegenerative disorders and something that must be corrected if neurons are to survive" says Dr. Ronald Davis from . New screening tech can help us find new ways to do this!

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  20. 19. sij
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