Ok, the Longevity FAQ will be released in a few minutes, wait for it
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methylations of CpGs versus young animals) look similar to a human 95% through its long lifespan? 2) What is known about the damage-restoration matrix: if I repair damage type X, does it automatically repair/restore damage type Y? For the various "hallmarks" and more specific
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subdivisions of damage types. Maybe most importantly, per Laura, if we reprogram epigenetics w/ Yamanaka type factors, which other types of damage automatically go away, and which don't? I feel like if I understood the answers to those, I'd have understood something about aging.
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I mostly focused on damage prevention vs damage repair as the literature is mostly the former, but I'm leaving these Q's in the further questions section. But I can say that there are kinds of damage that are relevant to us but not other animals, e.g. senescence
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I hope there is something (the table on animal models) in section 3.1 that answers a tiny % of what you had in mind
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