Ok, the Longevity FAQ will be released in a few minutes, wait for it
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model organisms like C. elegans, broadly related to some axis of growth/use energy now versus protect/conserve/repair for the future. But when we think about the various "hallmarks" or types of damage in aging, to what extent do different organisms share primary damage mechanisms
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, and can this be quantified. For example, if C. elegans is 95% through its short lifespan, does its per-cell lipofucin content, probability of senescence, or absolute degree of epigenetic deregulation (e.g., differences in states of histones or expression levels of genes or
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